Life Span:
The human organism has a fixed life span.
This genetically determined, species-specific constant is best as a fixed
number of normal cell replications. Once this normal number of cell cycles
reached, abnormal changes within the cell occur with increasing frequency.
Further the amount of time between cell division increases.
Life Expectancy:
Life
expectancy connotes a far different concept. It deals more closely with the
influence of environmental factors on the organism. Hostile environmental
factors diminish life expectancy whereas a conductive environment allows life
expectancy to genetically approach the determined life span. As the life
expectancy approaches life span in our society, quality of life issues become
increasingly important. It is with these issues that the physical caring for
the aging athlete must contend.
Aging Process:
Skeletal changes-
The aging process is associated with
predictable physiologic and biomechanical changes:
- Decrease in bone mass.
- Decrease in cortical thickness.
- Increase in cross sectional diameter.
A decrease in bone mass can be measured in
the normal human female by the time she reaches her mid-forties. Similarly,
normal males show a measurable decrease in bone mass by the time they reach
their fifties.
Factors that stimulates bone resorption
& bone loss:
•
low
serum calcium
•
high
serum phosphate
•
inactivity
(leads to disuse atrophy)
•
acidosis
•
steroids
The physiologic activity of bone throughout
life is subject to a variety of imbalances. As a general rule, the normal aging
process of males relates to a decrease in formation of bone in latter decades
of life. Women, however, are subject to increased resorption of bone,
particularly in the years surrounding menopause.
Factors that
slow bone resorption & bone loss:
•
estrogen
•
alkalosis
•
high
ratio of serum calcium to phosphorus
•
increased
activity level
•
high
clcium intake
Minimum adult requirement for dietary calcium:
·
Premenopausal
women – 1000 mg/day
·
Postmenopausal
women – 1500 mg/day
·
Lactating
women – 2000 mg/day
·
Men –
1000 mg/day
To a certain extent, loss of bone mass is reversible. An increase in
the level of activity, medication and dietary correction stimulates a
measurable increase in bone mass in all age group.
Soft tissue changes:
At
all ages myotendinous junction is the weakest link in the muscle tendon unit
and in its attachment to bone. Although the muscle tissue remains relatively
homogenous and fails at an unpredictable location. “Disuse atrophy” diminishes
the efficiency of the myotendinous unit and contributes to a loss of stiffness
and to a decreased load to failure.
Physiological changes in Aging muscle:
·
A loss
of motor units.
·
Decrease
in number of muscle fibres.
·
Decrease
in speed of contraction.
·
Increase
in reflex time.
·
Slower
relaxation phase of contraction.
·
Increased
average collagen fibril size in tendon.
·
Increased
insoluble collagen content and collagen cross linking in tendon (causes
increased stiffness of the aging tendon).
·
Decreased
tendon water, chondroitin sulphate and hyaluronic acid content.
·
Decreased
capillary density.
Healing response:
Vascular insufficiency, diabetes and
a variety of other disease process results in delayed healing. The literature
describing age related changes in skeletal and soft tissues has at least one
major flaw. All studies even those that stratify patients according to age,
fail to differentiate activity levels.
please post your references............
ReplyDelete